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66. Kindlon, T. (2017), ‘Do Graded Activity Therapies Cause Harm in Chronic Fatigue Syndrome?’ https://pubmed.ncbi.nlm.nih.gov/28805516/.
67. Taylor, A.K., et al. (2017), ‘“It’s personal to me”: A qualitative study of depression in young people with CFS/ME’: https://pubmed.ncbi.nlm.nih.gov/27742756/.
68. Kiecolt-Glaser, J.K., et al. (1986), ‘Modulation of Cellular Immunity in Medical Students’: http://pni.osumc.edu/KG Publications %28pdf%29/013.pdf.
69. Gouin, J-P. and Kiecolt-Glaser, J.K. (2011), ‘The impact of psychological stress on wound healing: methods and mechanisms’: www.ncbi.nlm.nih.gov/pubmed/ 21094925.
70. Kiecolt-Glaser, J.K., et al. (1995), ‘Slowing of wound healing by psychological stress’: https://pubmed.ncbi.nlm.nih.gov/7475659/.
71. Naviaux, R.K. (2014), ‘Metabolic features of the cell danger response’: www.sciencedirect.com/science/article/pii/S1567724913002390.
72. Naviaux, R.K. (2019), ‘Perspective: Cell Danger Response Biology – The New Science that Connects Environmental Health with Mitochondria and the Rising Tide of Chronic Illness’: www.sciencedirect.com/science/article/pii/S1567724919302922? via%3Dihub.
73. Naviaux, R.K., et al. (2016), ‘Metabolic features of chronic fatigue syndrome’: https://pubmed.ncbi.nlm.nih.gov/27573827/.
Глава 5. Новая модель понимания усталости
74. Belcourt, S., et al. (2001), ‘A twin Study of Chronic Fatigue’: www.academia.edu/ 2725172/.
75. Perez, M., et al. (2019), ‘Genetic Predisposition for Immune System, Hormone, and Metabolic Dysfunction in Myalgic Encephalomyelitis/Chronic Fatigue Syndrome: A Pilot Study’: www.ncbi.nlm.nih.gov/pmc/articles/PMC6542994/.
76. Fitzgerald, K. (not yet published), ‘Methylation Diet and Lifestyle Study’: https://clinicaltrials.gov/ct2/show/study/NCT03472820.
Глава 6. Личностные паттерны, вызывающие усталость
77. Tack, M. (2019), ‘Letter to the Editor (March 3, 2019) concerning the paper “The relationship between chronic fatigue syndrome, burnout, job satisfaction, social support and age among academics at a tertiary institution”’: www.journalssystem.com/ijomeh/Letter-tothe-editor-concerning-the-paper-The-relationship-between-chronicfatigue,105960,0,2.html.
78. Alderman, C. (1990), ‘Yuppie ’flu – a real illness’: http://journals.rcni.com/doi/ 10.7748/ns.4.49.18.s33.
Глава 7. Воздействие нагрузок на ваше тело
79. Felitti, V.J., et al. (2019), ‘Reprint of: Relationship of Childhood Abuse and Household Dysfunction to Many of the Leading Causes of Death in Adults: The Adverse Childhood Experiences (ACE) Study’: https://linkinghub.elsevier.com/retrieve/pii/S0749379719301436.
80. Finkelhor, D., et al. (2015), ‘A revised inventory of Adverse Childhood Experiences’: www.sciencedirect.com/science/article/abs/pii/S0145213415002409?via%3Dihub.
81. Afifi, T.O., etal. (2020), ‘Confirmatory factor analysis of adverse childhood experiences (ACEs) among a community-based sample of parents and adolescents’: www.ncbi.nlm.nih.gov/pubmed/32316954BMC.
82. Shalev, I., et al. (2020), ‘Investigating the impact of early-life adversity on physiological, immune, and gene expression responses to acute stress: A pilot feasibility study’: www.ncbi.nlm.nih.gov/pmc/articles/PMC7122782/.
83. Gerritsen, L., et al. (2017), ‘HPA Axis Genes, and Their Interaction with Childhood Maltreatment, are Related to Cortisol Levels and Stress-Related Phenotypes’: www.ncbi.nlm.nih.gov/pubmed/28589964 2017.
84. Steenkamp, M.M., et al. (2017), ‘Predictors of PTSD 40 years after combat: Findings from the National Vietnam Veterans longitudinal study’: http://doi.wiley.com/10.1002/da.22628.
85. Howley, E. (2019), ‘Statistics on PTSD in Veterans’: https://health.usnews.com/conditions/mental-health/ptsd/articles/ptsd-veteransstatistics.
Глава 8. Находитесь ли вы в состоянии исцеления?
86. Chu, B., et al. (2020), ‘Physiology, Stress Reaction’: www.ncbi.nlm.nih.gov/pubmed/ 31082164.
87. Porges, S.W. (2001), ‘The polyvagal theory: phylogenetic supstrates of a social nervous system’: www.sciencedirect.com/science/article/abs/pii/S0167876001001623? via%3Dihub.
88. Porges, S.W. (2009), ‘The polyvagal theory: new insights into adaptive reactions of the autonomic nervous system’: www.ncbi.nlm.nih.gov/pubmed/19376991.
89. Zimmermann, A., et al. (2014), ‘When less is more: hormesis against stress and disease’: http://microbialcell.com/researcharticles/when-lessis-more-hormesis- against-stress-and-disease/.
90. van Oort, J., et al. (2020), ‘Absence of default mode downregulation in response to a mild psychological stressor marks stress-vulnerability across diverse psychiatric disorders’: www.sciencedirect.com/science/article/pii/S2213158220300152.
91. Kinlein, S.A., et al. (2015), ‘Dysregulated hypothalamic-pituitaryadrenal axis function contributes to altered endocrine and neurobehavioral responses to acute stress’: www.ncbi.nlm.nih.gov/pubmed/25821436.
92. Dunlavey, C.J. (2018), ‘Introduction to the HypothalamicPituitary-Adrenal Axis: Healthy and Dysregulated Stress Responses, Developmental Stress and Neurodegeneration’: www.ncbi.nlm.nih.gov/pubmed/30057514.
Глава 9. Пищеварительная система и источники энергии
93. Sender, R., et al. (2016), ‘Revised Estimates for the Number of Human and Bacteria Cells in the Body’: www.ncbi.nlm.nih.gov/pmc/articles/PMC4991899/.
94. Belkaid, Y. and Hand, T.W. (2014), ‘Role of the microbiota in immunity and inflammation’: www.ncbi.nlm.nih.gov/pubmed/24679531.
95. Carabotti, M., et al. (2015), ‘The gut – brain axis: interactions between enteric microbiota, central and enteric nervous systems’: www.ncbi.nlm.nih.gov/pubmed/ 25830558.
96. Ma, Q., et al. (2019), ‘Impact of microbiota on central nervous system and neurological diseases: the gut – brain axis’: https://jneuroinflammation.biomed.central.com/articles/10.1186/s12974-019-1434-3.
97. Bischoff, S.C., et al. (2014), ‘Intestinal permeability – a new target for disease prevention and therapy’: www.ncbi.nlm.nih.gov/pmc/articles/PMC4253991/.
98. Li, Y, et al. (2018), ‘The Role of Microbiome in Insomnia, Circadian Disturbance and Depression’, Frontiers in Psychiatry, 9(December):1–11.
99. Appleton, J. (2018), ‘The Gut – Brain Axis: Influence of Microbiota on Mood and Mental Health’: www.ncbi.nlm.nih.gov/pmc/articles/PMC6469458/.
100. D’Mello, C., et al. (2015), ‘Probiotics Improve Inflammation-Associated Sickness Behavior by Altering Communication between the Peripheral Immune System and the Brain’: www.jneurosci.org/content/35/30/10821.full.
101. Ganci, M., et al. (2019), ‘The role of the brain – gut – microbiota axis in psychology: The importance of considering gut microbiota in the development, perpetuation, and treatment of psychological disorders’: www.ncbi.nlm.nih.gov/pmc/articles/PMC6851798/.
102. Parkar, S.G., et al. (2019), ‘Potential role for the gut microbiota in modulating host circadian rhythms and metabolic health’, Microorganisms, 7(2):1–21.
103. Fasano, A. (2020), ‘All disease begins in the (leaky) gut: role of zonulin-mediated gut permeability in the pathogenesis of some chronic inflammatory diseases’: www.ncbi.nlm.nih.gov/pubmed/32051759F1000.
104. Kolacz, J. and Porges, S.W. (2018), ‘Chronic Diffuse Pain and Functional Gastrointestinal Disorders After Traumatic Stress: Pathophysiology Through a Polyvagal Perspective’: www.ncbi.nlm.nih.gov/pubmed/29904631.
105. Maes, M. and Twisk, F.N. (2009), ‘Why myalgic encephalomyelitis/chronic fatigue syndrome (ME/CFS) may kill you: disorders in the inflammatory and oxidative and nitrosative stress (IO&NS) pathways may explain cardiovascular disorders in ME/CFS’: www.ncbi.nlm.nih.gov/pubmed/20038921.
106. Maes, M. (2009), ‘Inflammatory and oxidative and nitrosative stress pathways underpinning chronic fatigue, somatization and psychosomatic symptoms’: www.ncbi.nlm.nih.gov/pubmed/19127706.
107. Yu, LC-H. (2018), ‘Microbiota dysbiosis and barrier dysfunction in inflammatory bowel disease and colorectal cancers: exploring a common ground hypothesis’: www.ncbi.nlm.nih.gov/pmc/articles/PMC6234774/.
108. Bures, J., et al. (2006), ‘Small intestinal bacterial overgrowth syndrome’, World Journal of Gastroenterology, 16(24):2978–90.
109. Saffouri, G.B., etal. (2019), ‘Small intestinal microbial dysbiosis underlies symptoms associated with functional gastrointestinal disorders’: www.ncbi.nlm.nih.gov/ pmc/articles/PMC6494866/.
110. Sachdev, A.H. and Pimentel, M. (2013), ‘Gastrointestinal bacterial overgrowth: pathogenesis and clinical significance’: www.ncbi.nlm.nih.gov/pubmed/ 23997926.
111. Pimentel, M., et al. (2004), ‘A link between irritable bowel syndrome and fibromyalgia may be related to findings on lactulose breath testing’: www.ncbi.nlm.nih.gov/pubmed/15020342.
Глава 10. Гормоны и резервная энергетическая система тела
112. Cadegiani, F.A. and Kater, C.E. (2016), ‘Adrenal fatigue does not exist: a systematic review’: www.ncbi.nlm.nih.gov/pmc/articles/PMC4997656/.
113. Wyller, V.B., et al. (2016), ‘Altered neuroendocrine control and association to clinical symptoms in adolescent chronic fatigue syndrome: a cross-sectional study’: www.ncbi.nlm.nih.gov/pubmed/27149955.
114. Torres-Harding, S., et al. (2008), ‘The associations between basal salivary cortisol and illness symptomatology in chronic fatigue syndrome’: www.ncbi.nlm.nih.gov/pmc/articles/PMC2730359/.
115. Ludwig, D.S. (2014), ‘Clinical update: the low-glycaemic-index diet. https://www.thelancet.com/journals/lancet/article/PIIS01406736%2807%2960427-9/fulltext.
116. Savage, D.B., et al. (2007), ‘Disordered Lipid Metabolism and the Pathogenesis of Insulin Resistance’: www.ncbi.nlm.nih.gov/pmc/articles/PMC2995548/.
117. Moghaddam, E., et al. (2006), ‘The Effects of Fat and Protein on Glycemic Responses in Nondiabetic Humans Vary with Waist Circumference, Fasting Plasma Insulin, and Dietary Fiber Intake’: https://academic.oup.com/jn/article/136/10/ 2506/4746688.
118. Hucklebridge, F., et al. (2005), ‘The diurnal patterns of the adrenal steroids cortisol and dehydroepiandrosterone (DHEA) in relation
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