Читать книгу - "Ваш мозг. Что нейронаука знает о мозге и его причудах - Марк Дингман"

^1. A. Dolan, “Always Smiling, the Stroke Patient Who Can’t Feel Sad,” The Daily Mail, August 12, 2013, Health, http://www.dailymail. co.uk/health/article-2389891/Always-smiling-stroke-patient-feel-sad-Condition-leaves-Grandfather-permanently-happy-prone-fits-giggles-inappropriate-times.html.

^2. D.J. Felleman and D.C. Van Essen, “Distributed Hierarchical Processing in the Primate Cerebral Cortex,” Cerebral Cortex 1, no. 1 (1991): 1-47.

^3. P. Broca, “Comparative Anatomy of the Cerebral Convolutions: The Great Limbic Lobe and the Limbic Fissure in the Mammalian Series,” Journal of Comparative Neurology 523, no. 17 (December 2015): 2501–2554.

^4. J.W Papez, “A Proposed Mechanism of Emotion,” Archives of Neurology and Psychiatry 38 (1937): 725–743.

^5. P.D. MacLean, “Some Psychiatric Implications of Physiological Studies on Frontotemporal Portion of Limbic System (Visceral Brain),” Electroencephalography and Clinical Neurophysiology 4, no. 4 (November 1952): 407–418.

^6. M.S. George, T.A. Ketter, P.I. Parekh, B. Horwitz, P. Herscovitch, and R.M. Post, “Brain Activity during Transient Sadness and Happiness in Healthy Women,” American Journal of Psychiatry 152, no. 3 (March 1995): 341–351.

^7. H.S. Mayberg, M. Liotti, S.K. Brannan, S. McGinnis, R.K. Mahurin, P.A. Jerabek, J.A. Silva, et al., “Reciprocal Limbic-Cortical Function and Negative Mood: Converging PET Findings in Depression and Normal Sadness,” American Journal of Psychiatry 156, no. 5 (May 1999): 675–682.

^8. P.E. Greenberg, R.C. Kessler, H.G. Birnbaum, S.A. Leong, S.W. Lowe, P.A. Berglund, and P.K. Corey-Lisle, “The Economic Burden of Depression in the United States: How Did It Change between 1990 and 2000?” Journal of Clinical Psychiatry 64, no. 12 (December 2003): 1465–1475.

^9. B. Voinov, WD. Richie, and R.K. Bailey, “Depression and Chronic Diseases: It Is Time for a Synergistic Mental Health and Primary Care Approach,” The Primary Care Companion for CNS Disorders 15, no. 2 (2013): PCC.12r01468.

^10. A. Mykletun, O. Bjerkeset, S. Overland, M. Prince, M. Dewey, and R. Stewart, “Levels of Anxiety and Depression as Predictors of Mortality: The HUNT Study,” British Journal of Psychiatry 195, no. 2 (August 2009): 118–125.

¹¹ E.A. Osuch, T.A. Ketter, T.A. Kimbrell, M.S. George, B.E. Benson, M.W. Willis, P. Herscovitch, and R.M. Post, “Regional Cerebral Metabolism Associated with Anxiety Symptoms in Affective Disorder Patients,” Biological Psychiatry 48, no. 10 (November 2000): 1020–1023.

^12. Mayberg et al., “Reciprocal Limbic-Cortical Function and Negative Mood,” 675–682.

^13. T. Hajek, J. Kozeny, M. Kopecek, M. Alda, and C. Hoschl, “Reduced Subgenual Cingulate Volumes in Mood Disorders: A MetaAnalysis,” Journal of Psychiatry & Neuroscience 33, no. 2 (March 2008): 91–99.

^14. D.L. Dunner, A.J. Rush, J.M. Russell, M. Burke, S. Woodard, P. Wingard, and J. Allen, “Prospective, Long-Term, Multicenter Study of the Naturalistic Outcomes of Patients with TreatmentResistant Depression,” Journal of Clinical Psychiatry 67 (2006): 688–695.

^15. M.T. Berlim, A. McGirr, F. Van den Eynde, M.P. Fleck, and P. Giacobbe, “Effectiveness and Acceptability of Deep Brain Stimulation (DBS) of the Subgenual Cingulate Cortex for TreatmentResistant Depression: A Systematic Review and Exploratory Meta-Analysis,” Journal of Affective Disorders 159 (April 2014): 31–38.

^16. K.S. Choi, P. Riva-Posse, R.E. Gross, and H.S. Mayberg, “Mapping the ‘Depression Switch’ during Intraoperative Testing of Subcallosal Cingulate Deep Brain Stimulation,” JAMA Neurology 72, no. 11 (November 2015): 1252–1260.

^17. Там же.

^18. B.H. Bewernick, S. Kayser, V. Sturm, and T.E. Schlaepfer, “LongTerm Effects of Nucleus Accumbens Deep Brain Stimulation in Treatment-Resistant Depression: Evidence for Sustained Efficacy,” Neuropsychopharmacology 37, no. 9 (August 2012): 1975–1985.

^19. J.L. Price and W.C. Drevets, “Neural Circuits Underlying the Pathophysiology of Mood Disorders,” Trends in Cognitive Sciences 16, no. 1 (January 2012): 61–71.

^20. G.M. Cooney, K. Dwan, C.A. Greig, D.A. Lawlor, J. Rimer, ER. Waugh, M. McMurdo, and G.E. Mead, “Exercise for Depression,” The Cochrane Database of Systematic Reviews 12, no. 9 (September 2013): CD004366.

²¹ Цит. по: F. Lopez-Munoz and C. Alamo, “Monoaminergic Neurotransmission: The History of the Discovery of Antidepressants from 1950s Until Today,” Current Pharmaceutical Design 15 (2009): 1563–1586.

^22. E. Shorter, A History of Psychiatry: From the Era of the Asylum to the Age of Prozac (New York: John Wiley & Sons, Inc., 1997).

^23. National Center for Health Statistics, “Health, United States, 2010: With Special Feature on Death and Dying,” 2011 Feb. Report, 2011.

^24. P.D. Kramer, Listening to Prozac: A Psychiatrist Explores Antidepressant Drugs and the Remaking of the Self (New York: Penguin Books, 1993).

^25. R. Invernizzi, C. Velasco, M. Bramante, A. Longo, and R. Samanin, “Effect of 5-HT1A Receptor Antagonists on Citalopram-Induced Increase in Extracellular Serotonin in the Frontal Cortex, Striatum and Dorsal Hippocampus,” Neuropharmacology 36, no. 4–5 (1997): 467–473.

^26. G.R. Heninger, P.L. Delgado, and D.S. Charney, “The Revised Monoamine Theory of Depression: A Modulatory Role for Monoamines, Based on New Findings from Monoamine Depletion Experiments in Humans,” Pharmacopsychiatry 29, no. 1 (1996): 2-11.

^27. I. Kirsch, B.J. Deacon, T.B. Huedo-Medina, A. Scoboria, T.J. Moore, and B.T. Johnson, “Initial Severity and Antidepressant Benefits: A Meta-Analysis of Data Submitted to the Food and Drug Administration,” PLoS Medicine 5, no. 2 (2008): e4. Важно отметить, что данная тема неоднозначна и работа авторов была во многом раскритикована. Результаты исследования были опубликованы в 2008 г., и впоследствии появились свидетельства как в поддержку, так и в опровержение полученных в его ходе результатов. Даже в тех исследованиях, которые подтверждают эффективность антидепрессантов, уточняется, что их воздействие на пациента очень ограничено.